Too many potatoes could trigger diabetes

Whether you say “potayto” or “potahto,” the latest research says they are a no-no if you want to stay healthy.

According to a new study, published in Diabetes Care, potatoes don’t just pack on the belly fat — they can also significantly raise the risk of type 2 diabetes.

Researchers at the Harvard School of Public Health reviewed information from three studies of nearly 50,000 healthy doctors and nurses without diabetes. Their results showed that eating one serving of potatoes a day for four years increased the risk of the disease by 33 per cent. Even eating a fraction of that — just two to four portions of potatoes a week — increased the risk by 7 per cent.

French fries were the real culprit. For every three servings of French fries people ate per week, they raised their risk by a whopping 19 per cent, compared to those who ate baked, boiled, or mashed potatoes.

The thing about potatoes is that even though we consider them a “vegetable” they actually contain a large amount of starch and not much fibre, vitamins, minerals, or the disease-fighting compounds found in most other fruits and veggies.

And when they’re served hot, the starch becomes easier to digest… easier to get into your bloodstream… and easier to raise your blood glucose levels.

The truth is most people think that sugar is the only problem for people with diabetes — but starch is also problematic, because of the way your body breaks it down into sugar (glucose).

The study suggests replacing potatoes with whole grains. But I say skip the starches altogether. Ditch the home fries and bulk up on steak and eggs for breakfast.

And if you’re really missing your mashed potatoes, try substituting white potatoes with Paleo-friendly sweet potatoes, or true root vegetables like turnips and parsnips. You’ll get a similar mouth feel with a lot more fibre, nutrients, and even flavour.

Wishing you the best of health,

Dr. Glenn S. Rothfeld
Nutrition & Healing
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“Too many potatoes could trigger diabetes,” The Daily Mail, Dec. 30, 2015

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